=[Yes, the] Olsen twins create nutrition show - [AND it makes the wire in the UK!]
Monday July 3, 11:45 AM - By WENN
Click to enlarge photo -[your choice]
Mary-Kate and Ashley Olsen are creating a show about health and nutrition for young children, according to media reports in the US.
Ashley and her twin sister Mary-Kate - who was underwent rehab for anorexia in 2004 - have met with US TV executives about the new program aimed at four-to-five-year-olds called Sportee Kids.
A source tells American publication Us Weekly, "It will focus on fitness and healthy eating."
The duo have reportedly received a mixed reactions about their new concept.
The source adds, "Some execs are weirded out to do a show about food and nutrition with the Olsens."
A representative for the pair has hit back saying, "There's no truth to people being scared to work with them. They're healthy and fine." [The twins' regular publicist is Michael Pagnotta].
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Wednesday, July 05, 2006
Monday, July 03, 2006
Yes, Star Jones Reynolds has left The View - and another Media Feeding Frenzy begins --or---slooooow newz day - hit snooze button NOW
fox: http://www.foxnews.com/story/0,2933,201940,00.html jane hall, gabler, cal thomas, the usual suspects
MSNBC - just google it anywhere
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MSNBC - just google it anywhere
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Jossip editors, oh, where out thou NOW?
Good! SHAKE IT UP! Do we need new blood in the mix, or should we care?FILE UNDER - mediabistro reports to the "insiders"
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i need a martini, jerk! hurry up!
Sunday, July 02, 2006
The "I can get sued" Dept. - No, I can't! Star Jones (already) on the comeback trail - not a publicity stunt - I AM THE VICTIM HERE
http://goodpublicitybadpublicity.blogspot.com/2006/07/star-jones-and-al-roker-rebuilding.html#links
http://video.msn.com/v/us/msnbc.htm?g=FAB77296-31CF-4B4A-BE9C-B52F4A9C5ED6&f=00&fg=copy
Star Jones is the ultimate DIVA - love her, hate her, she is a S T A R. But now she is out of the closet - she 'fessed up to Al Roker on TODAY. Bigger than Larry King which was a benchwarmer. It was long segment -- Longer than "Hillary Clinton and the vast right wing conspiracy" (!) It will take one hell of a brand reshaping here, But Savvy Star is getting busy so she lands another gig NOW. She is taking the high road.
Hindsight is 20 - 15: Yes, she should have had the public join the journey at Day #1. Just a purely PR opine, natch. But this is a very good route. Confession to Al Roker. Good move. Apologized to viewers (ala DeGeneres) Better move. And if a "Medical Intervention" is the bypass, well, alrighty, then! (She and Al had the same doctors, after all). [By the way, I know Dr. Paul Lin personally--he almost did a re-do for me!]
Good luck, Mrs. Reynolds! Be ready: Barbara Walters will speak OUT sooner or later!
Abbe B., -92 now. Sans bypass and redo of a roux -n-y.
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"The Obesity Tsunami": --or--REASONS for morbid obesity other than food are:
AFP: Chewing the fat: New theories on world's obesity pandemicJul 01 11:33 PM US/Eastern
Fatty hamburgers, sugar-laden sodas and a couch-potato lifestyle: these are the familiar villains in the crisis of obesity sweeping developed countries.
But what if they had been convicted without fair trial?
What if the global fat explosion had other causes?
What, for instance, if air conditioning or lack of sleep helped make you fat? Or what if obesity were caused by a microbe -- what if, bang, you caught an unlucky sneeze and this made you chub out?
These ideas challenge the mainstream view that the bulging waistlines of an advancing society can be overwhelmingly pinned to diet and lifestyle.
The World Health Organisation (WHO) last September warned that a billion people were overweight and obese, and the toll could rise to 1.5 billion by 2015, driven by low- and middle-income countries.
The WHO accepted there were "a number of factors" for this increase, but especially blamed "a global shift in diet towards increased energy, fat, salt and sugar intake, and a trend towards decreased physical activity due to the sedentary nature of modern work and transportation, and increasing urbanisation."
Some worry that this view is dangerously monolithic.
Writing on Tuesday in the International Journal of Obesity, a team of US public-health experts caution against focussing obsessively on the "Big Two" -- a slower lifestyle and modern food marketing.
"This has created a hegemony whereby the importance of the Big Two is accepted as established and other putative factors are not seriously explored," they say. "The result may be well-intentioned but ill-founded proposals for reducing obesity rates."
They contend the evidence against junk food, supersize-me portions and high-calorie corn syrup is "equivocal and largely circumstantial" and offer some intriguing ideas of their own for other drivers of the obesity tsunami.
Among them:
-- Industrial chemicals called endocrine disruptors that disturb metabolism, encouraging the formation of fat.
-- Giving up smoking: people who give up cigarettes very often gain weight.
-- Air conditioning, which establishes a comfortable temperature zone. In temperatures above this zone, people eat less. The rise in number of air-conditioned homes in the United States virtually mirrors the increase in the US obesity rate.
-- Fat people marry other fat people. These individuals may be genetically vulnerable to obesity, a trait that could handed on to their children.
Another hypothesis is that lack of sleep jolts the metabolic system into demanding doses of instant energy. SUGAR, ANYONE?
University of Chicago researcher Esra Tasali notes that waistlines in modern societies started to expand when people started to sleep less. Today, the "sleep deficit" is about two hours per night compared with 40 years ago.
In work unveiled at an obesity conference last October, Tasali recruited a group of healthy young adults and divided them into three groups. One group had eight hours' sleep; another had their sleep regime extended to 12 hours; and the third was limited to only four hours.
The sleep-deprived group swiftly developed cravings for high-calorie sweets, and their metabolisms were akin to those of diabetics.
Meanwhile, Nikhil Dhurandhar of the Pennington Biomedical Research Center at Louisiana State University believes obesity could be caused by a bug.
At least 10 different pathogens are known to cause obesity in animals, causing dramatic changes to the metabolic system so that more energy gets converted into fat.
Dhurandhar believes that something similar may happen among humans exposed to cousins of the common cold.
He tested the stored blood of 500 Americans and found that 30 percent of obese people had antibodies for Ad-36, an adenovirus which causes coughing, sneezing and cold-like symptoms.
Only 11 percent of people of normal body weight had this telltale of Ad-36 infection.
Dhurandhar stresses, though, that infection is likely to be only of a bouquet of causes for obesity.
"In 10 years, people may be able to walk into a clinic and be told that their obesity is due to X cause, such as genes, the endocrine system or pathogens. That may have a more productive outcome than a blanket treatment right now, (which) is not very successful."
Neville Rigby, of the European Association for the Study of Obesity, says that such unconventional views usually get a good hearing among scientists, for no one claims to have a monopoly of wisdom when it comes to this fast-growing disease.
"It's a very complex story, it's not a single issue," said Rigby. "But the overarching question is how much we consume and how much we burn."
Copyright AFP 2006
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Fatty hamburgers, sugar-laden sodas and a couch-potato lifestyle: these are the familiar villains in the crisis of obesity sweeping developed countries.
But what if they had been convicted without fair trial?
What if the global fat explosion had other causes?
What, for instance, if air conditioning or lack of sleep helped make you fat? Or what if obesity were caused by a microbe -- what if, bang, you caught an unlucky sneeze and this made you chub out?
These ideas challenge the mainstream view that the bulging waistlines of an advancing society can be overwhelmingly pinned to diet and lifestyle.
The World Health Organisation (WHO) last September warned that a billion people were overweight and obese, and the toll could rise to 1.5 billion by 2015, driven by low- and middle-income countries.
The WHO accepted there were "a number of factors" for this increase, but especially blamed "a global shift in diet towards increased energy, fat, salt and sugar intake, and a trend towards decreased physical activity due to the sedentary nature of modern work and transportation, and increasing urbanisation."
Some worry that this view is dangerously monolithic.
Writing on Tuesday in the International Journal of Obesity, a team of US public-health experts caution against focussing obsessively on the "Big Two" -- a slower lifestyle and modern food marketing.
"This has created a hegemony whereby the importance of the Big Two is accepted as established and other putative factors are not seriously explored," they say. "The result may be well-intentioned but ill-founded proposals for reducing obesity rates."
They contend the evidence against junk food, supersize-me portions and high-calorie corn syrup is "equivocal and largely circumstantial" and offer some intriguing ideas of their own for other drivers of the obesity tsunami.
Among them:
-- Industrial chemicals called endocrine disruptors that disturb metabolism, encouraging the formation of fat.
-- Giving up smoking: people who give up cigarettes very often gain weight.
-- Air conditioning, which establishes a comfortable temperature zone. In temperatures above this zone, people eat less. The rise in number of air-conditioned homes in the United States virtually mirrors the increase in the US obesity rate.
-- Fat people marry other fat people. These individuals may be genetically vulnerable to obesity, a trait that could handed on to their children.
Another hypothesis is that lack of sleep jolts the metabolic system into demanding doses of instant energy. SUGAR, ANYONE?
University of Chicago researcher Esra Tasali notes that waistlines in modern societies started to expand when people started to sleep less. Today, the "sleep deficit" is about two hours per night compared with 40 years ago.
In work unveiled at an obesity conference last October, Tasali recruited a group of healthy young adults and divided them into three groups. One group had eight hours' sleep; another had their sleep regime extended to 12 hours; and the third was limited to only four hours.
The sleep-deprived group swiftly developed cravings for high-calorie sweets, and their metabolisms were akin to those of diabetics.
Meanwhile, Nikhil Dhurandhar of the Pennington Biomedical Research Center at Louisiana State University believes obesity could be caused by a bug.
At least 10 different pathogens are known to cause obesity in animals, causing dramatic changes to the metabolic system so that more energy gets converted into fat.
Dhurandhar believes that something similar may happen among humans exposed to cousins of the common cold.
He tested the stored blood of 500 Americans and found that 30 percent of obese people had antibodies for Ad-36, an adenovirus which causes coughing, sneezing and cold-like symptoms.
Only 11 percent of people of normal body weight had this telltale of Ad-36 infection.
Dhurandhar stresses, though, that infection is likely to be only of a bouquet of causes for obesity.
"In 10 years, people may be able to walk into a clinic and be told that their obesity is due to X cause, such as genes, the endocrine system or pathogens. That may have a more productive outcome than a blanket treatment right now, (which) is not very successful."
Neville Rigby, of the European Association for the Study of Obesity, says that such unconventional views usually get a good hearing among scientists, for no one claims to have a monopoly of wisdom when it comes to this fast-growing disease.
"It's a very complex story, it's not a single issue," said Rigby. "But the overarching question is how much we consume and how much we burn."
Copyright AFP 2006
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